pdtc中文

• 二硫氨基甲酸肽吡咯烷
• 二硫代氨基甲酸吡咯烷
• 抑制劑吡咯烷二硫氨基甲酸
• 吡咯烷二硫氨基甲酸酯
+更多解釋...
• 啉烷二甲基硫脲

例句與用法

1. There are still many questions remain unanswered , including the mechanisms that specify oligochitosan recognition , the participation of additional molecules in transmembrane signaling , and the repertoire of downstream pathways activated by oligochitosan . it was also observed that pdtc did not totally block no production , which gave us another evidence that there must be some other signal transduction pathways ( perhaps mapk , pkc , ca2 + dependent signaling ) involved in the activation , so further investigation is necessary to clarify molecular mechanism of oligochitosan inducing immunological en
   Westemblot結果顯示100林歲ml劑量的殼寡糖作用于raw264 . 7細胞( 4x105個細胞/ ml ) 4h ，胞核內p65的含量明顯增加， 6h時達到高峰， 10h時恢復到基礎表達量，這就提示在殼寡糖對巨噬細胞的激活過程中nf一kb活化狀態可以持續6個小時。
2. The protein level of nf - kb in nucleus peaks at 6 h after stimulation and persists for at least 6 h . pdtc ( pyrrolidinedithiocarbamate ) , an inhibitor of nf - kb , the effective concentration of it for nf - kb inhibition without any toxicity on cells is up to 100 umol / l , can significantly block no production induced by oligochitosan . although the study reported herein reveals that oligochitosan induces no production via nf - kb in macrophage , which is an important signaling mechanism of oligochitosan inducing immuno - enhancement of macrophage in vitro
   加入100林mol / l劑量的nf一忍抑制劑毗咯烷二硫代氨基甲酸鹽( pnte )作用于rawz “ . 7細胞( 4火10 ，個細胞/ ml ) 90rnin ，然后再加入100林留而劑量的殼寡糖對巨噬細胞進行刺激，發現pdtc在各個時間點( 6 、 12 、 18 、 24h )都明顯抑制殼寡糖對巨噬細胞no的誘導作用。