cell adhesion molecules造句

• Prokaryotic expression of neural cell adhesion molecule l1 - ig and its function
  的原核表達及其功能
• Liver cell adhesion molecule , lcam
  肝細胞粘合分子
• Structure and action of neural cell adhesion molecules in nervous system
  神經細胞黏附分子結構及其在神經系統的作用
• Study progress in cell adhesion molecule and the pathogenesy of coronary heart disease
  粘附分子與冠心病發病機制研究進展
• The effects of xueshuanxinmaining on cell adhesion molecules in dyslipidemia patients
  血栓心脈寧對高脂血癥患者粘附分子的影響
• Expression and significance of the vascular cell adhesion molecule - 1 in cardiac allografts of rats
  1在大鼠心臟移植慢性排斥模型中的表達
• Study on relationship between soluble vascular cell adhesion molecule - 1 and senile multi - infarct dementia
  1與老年多梗死性癡呆關系的研究
• Variation of soluble vascular cell adhesion molecule - 1 in serum of patients with cerebral infarction
  腦梗死患者血清可溶性血管細胞黏附分子1的變化
• Effects of yiqi huayu recipe on neural cell adhesion molecule in rats with lumbar nerve root compression
  益氣化瘀方對腰神經根損傷大鼠神經細胞黏附分子的作用
• Catenin binds to the cytoplasmic tail of a transmembrane cell adhesion molecule - epithelial cadherin ( e - cadherin ) in epithelial cells and forms an adhesion complex [ e - cadherin - p - catenin ( or y - catenin ) - cc - catenin ] ( cadherin - catenin - complex , ccc ) , this complex play an important role in maintaining epithelial cell adhesion . some catenins such as - , - and p120ctn catenins are also crucial in cell signaling , they play an important role in cell proliferation and cell apoptosis
  在上皮細胞內，連環蛋白均能與跨膜細胞粘附分子?上皮鈣粘蛋白( e - cadherin )的胞內肽段結合而形成了由上皮鈣粘蛋白- -連環蛋白( -連環蛋白) - -連環蛋白組成的粘附連接體( adherensjunctions , ajs ) ，這種復合體對維持細胞正常的粘附功能是必需的。
• It's difficult to see cell adhesion molecules in a sentence. 用cell adhesion molecules造句挺難的
• Conclusion one mechanism of wmt on aso may be its inhibition on arteriosclerosis by way of downregulating the expression of vascular endothelial cells adhesion molecules to decrease the adhesion of monocyte to vec , therefore to inhibit the monocytes migrating into vascular intima to develop foam cells
  結論下調血管內皮細胞黏附分子的表達，減少單核細胞與內皮細胞的黏附，從而抑制單核細胞遷移至血管內膜形成泡沫細胞，抑制動脈粥樣硬化as的形成，可能是溫脈通治療aso的機制之一。
• Conclusion one mechanism of wmt on aso may be its inhibition on arteriosclerosis by way of downregulating the expression of vascular endothelial cells adhesion molecules to decrease the adhesion of monocyte to vec , therefore to inhibit the monocytes migrating into vascular intima to develop foam cells . 2007 vol
  結論下調血管內皮細胞黏附分子的表達，減少單核細胞與內皮細胞的黏附，從而抑制單核細胞遷移至血管內膜形成泡沫細胞，抑制動脈粥樣硬化as的形成，可能是溫脈通治療aso的機制之一。